case study kf

Support for the Working Memory Model

Shallice and warrington (1974) - study of kf.

The working memory model is supported by evidence from brain damaged patients such as KF.

Research aim & method

• Aim: To investigate a patient KF who had suffered brain damage in a motorcycle accident.
• Method: A case study using numerous psychometric tests, experiments and observations.

• KF’s short term memory problems were much greater for auditory information than visual, suggesting his brain damage was restricted to the phonological loop.

• The case of KF supports both the MSM and the WMM as his LTM was unaffected by his injury, suggesting LTM and STM are different stores.
• His case supports the WMM as his visuospatial sketchpad seems unaffected by his injury, suggesting that resides in a different area of the brain to the phonological loop which was damaged.

• In depth and detailed.
• Cannot generalise from a case study.

Limitations of the Working Memory Model

Here are some limitations of the working memory model.

Central executive

• What does the central executive actually do?
• The model suggests that it allocates attention, but it is not fully explained why it is needed.

Lacks ecological validity

• Dual task experiments are very artificial – lacks ecological validity.

Lack of generalisability

• Cannot generalise from case studies on brain damaged patients.

Ambiguity about the LTM

• It is only a model of working memory and leaves many unanswered questions about the structure of LTM.

1 Social Influence

1.1 Social Influence

1.1.1 Conformity

1.1.2 Asch (1951)

1.1.3 Sherif (1935)

1.1.4 Conformity to Social Roles

1.1.5 BBC Prison Study

1.1.6 End of Topic Test - Conformity

1.1.7 Obedience

1.1.8 Analysing Milgram's Experiment

1.1.9 Agentic State & Legitimate Authority

1.1.10 Variables of Obedience

1.1.11 Resistance to Social Influence

1.1.12 Minority Influence & Social Change

1.1.13 Minority Influence & Social Impact Theory

1.1.14 End of Topic Test - Social Influences

1.1.15 Exam-Style Question - Conformity

1.1.16 Top Grade AO2/AO3 - Social Influence

2.1.1 Multi-Store Model of Memory

2.1.2 Short-Term vs Long-Term Memory

2.1.3 Long-Term Memory

2.1.4 Support for the Multi-Store Model of Memory

2.1.5 Duration Studies

2.1.6 Capacity Studies

2.1.7 Coding Studies

2.1.8 The Working Memory Model

2.1.9 The Working Memory Model 2

2.1.10 Support for the Working Memory Model

2.1.11 Explanations for Forgetting

2.1.12 Studies on Interference

2.1.13 Cue-Dependent Forgetting

2.1.14 Eye Witness Testimony - Loftus & Palmer

2.1.15 Eye Witness Testimony Loftus

2.1.16 Eyewitness Testimony - Post-Event Discussion

2.1.17 Eyewitness Testimony - Age & Misleading Questions

2.1.18 Cognitive Interview

2.1.19 Cognitive Interview - Geiselman & Fisher

2.1.20 End of Topic Test - Memory

2.1.21 Exam-Style Question - Memory

2.1.22 A-A* (AO3/4) - Memory

3 Attachment

3.1 Attachment

3.1.1 Caregiver-Infant Interaction

3.1.2 Condon & Sander (1974)

3.1.3 Schaffer & Emerson (1964)

3.1.4 Multiple Attachments

3.1.5 Studies on the Role of the Father

3.1.6 Animal Studies of Attachment

3.1.7 Explanations of Attachment

3.1.8 Attachment Types - Strange Situation

3.1.9 Cultural Differences in Attachment

3.1.10 Disruption of Attachment

3.1.11 Disruption of Attachment - Privation

3.1.12 Overcoming the Effects of Disruption

3.1.13 The Effects of Institutionalisation

3.1.14 Early Attachment

3.1.15 Critical Period of Attachment

3.1.16 End of Topic Test - Attachment

3.1.17 Exam-Style Question - Attachment

3.1.18 Top Grade AO2/AO3 - Attachment

4 Psychopathology

4.1 Psychopathology

4.1.1 Definitions of Abnormality

4.1.2 Definitions of Abnormality 2

4.1.3 Phobias, Depression & OCD

4.1.4 Phobias: Behavioural Approach

4.1.5 Evaluation of Behavioural Explanations of Phobias

4.1.6 Depression: Cognitive Approach

4.1.7 OCD: Biological Approach

4.1.8 Evidence for the Biological Approach

4.1.9 End of Topic Test - Psychopathy

4.1.10 Exam-Style Question - Phobias

4.1.11 Top Grade AO2/AO3 - Psychopathology

5 Approaches in Psychology

5.1 Approaches in Psychology

5.1.1 Psychology as a Science

5.1.2 Origins of Psychology

5.1.3 Reductionism & Problems with Introspection

5.1.4 The Behaviourist Approach - Classical Conditioning

5.1.5 Pavlov's Experiment

5.1.6 Little Albert Study

5.1.7 The Behaviourist Approach - Operant Conditioning

5.1.8 Social Learning Theory

5.1.9 The Cognitive Approach 1

5.1.10 The Cognitive Approach 2

5.1.11 The Biological Approach

5.1.12 Gottesman (1991) - Twin Studies

5.1.13 Brain Scanning

5.1.14 Structure of Personality & Little Hans

5.1.15 The Psychodynamic Approach (A2 only)

5.1.16 Humanistic Psychology (A2 only)

5.1.17 Aronoff (1957) (A2 Only)

5.1.18 Rogers' Client-Centred Therapy (A2 only)

5.1.19 End of Topic Test - Approaches in Psychology

5.1.20 Exam-Style Question - Approaches in Psychology

5.2 Comparison of Approaches (A2 only)

5.2.1 Psychodynamic Approach

5.2.2 Cognitive Approach

5.2.3 Biological Approach

5.2.4 Behavioural Approach

5.2.5 End of Topic Test - Comparison of Approaches

6 Biopsychology

6.1 Biopsychology

6.1.1 Nervous System Divisions

6.1.2 Neuron Structure & Function

6.1.3 Neurotransmitters

6.1.4 Endocrine System Function

6.1.5 Fight or Flight Response

6.1.6 The Brain (A2 only)

6.1.7 Localisation of Brain Function (A2 only)

6.1.8 Studying the Brain (A2 only)

6.1.9 CIMT (A2 Only) & Postmortem Examinations

6.1.10 Biological Rhythms (A2 only)

6.1.11 Studies on Biological Rhythms (A2 Only)

6.1.12 End of Topic Test - Biopsychology

6.1.13 Top Grade AO2/AO3 - Biopsychology

7 Research Methods

7.1 Research Methods

7.1.1 Experimental Method

7.1.2 Observational Techniques

7.1.3 Covert, Overt & Controlled Observation

7.1.4 Self-Report Techniques

7.1.5 Correlations

7.1.6 Exam-Style Question - Research Methods

7.1.7 End of Topic Test - Research Methods

7.2 Scientific Processes

7.2.1 Aims, Hypotheses & Sampling

7.2.2 Pilot Studies & Design

7.2.3 Questionnaires

7.2.4 Variables & Control

7.2.5 Demand Characteristics & Investigator Effects

7.2.6 Ethics

7.2.7 Limitations of Ethical Guidelines

7.2.8 Consent & Protection from Harm Studies

7.2.9 Peer Review & The Economy

7.2.10 Validity (A2 only)

7.2.11 Reliability (A2 only)

7.2.12 Features of Science (A2 only)

7.2.13 Paradigms & Falsifiability (A2 only)

7.2.14 Scientific Report (A2 only)

7.2.15 Scientific Report 2 (A2 only)

7.2.16 End of Topic Test - Scientific Processes

7.3 Data Handling & Analysis

7.3.1 Types of Data

7.3.2 Descriptive Statistics

7.3.3 Correlation

7.3.4 Evaluation of Descriptive Statistics

7.3.5 Presentation & Display of Data

7.3.6 Levels of Measurement (A2 only)

7.3.7 Content Analysis (A2 only)

7.3.8 Case Studies (A2 only)

7.3.9 Thematic Analysis (A2 only)

7.3.10 End of Topic Test - Data Handling & Analysis

7.4 Inferential Testing

7.4.1 Introduction to Inferential Testing

7.4.2 Sign Test

7.4.3 Piaget Conservation Experiment

7.4.4 Non-Parametric Tests

8 Issues & Debates in Psychology (A2 only)

8.1 Issues & Debates in Psychology (A2 only)

8.1.1 Culture Bias

8.1.2 Sub-Culture Bias

8.1.3 Gender Bias

8.1.4 Ethnocentrism

8.1.5 Cross Cultural Research

8.1.6 Free Will & Determinism

8.1.7 Comparison of Free Will & Determinism

8.1.8 Reductionism & Holism

8.1.9 Reductionist & Holistic Approaches

8.1.10 Nature-Nurture Debate

8.1.11 Interactionist Approach

8.1.12 Nature-Nurture Methods

8.1.13 Nature-Nurture Approaches

8.1.14 Idiographic & Nomothetic Approaches

8.1.15 Socially Sensitive Research

8.1.16 End of Topic Test - Issues and Debates

9 Option 1: Relationships (A2 only)

9.1 Relationships: Sexual Relationships (A2 only)

9.1.1 Sexual Selection & Human Reproductive Behaviour

9.1.2 Intersexual & Intrasexual Selection

9.1.3 Evaluation of Sexual Selection Behaviour

9.1.4 Factors Affecting Attraction: Self-Disclosure

9.1.5 Evaluation of Self-Disclosure Theory

9.1.6 Self Disclosure in Computer Communication

9.1.7 Factors Affecting Attraction: Physical Attributes

9.1.8 Matching Hypothesis Studies

9.1.9 Factors Affecting Physical Attraction

9.1.10 Factors Affecting Attraction: Filter Theory 1

9.1.11 Factors Affecting Attraction: Filter Theory 2

9.1.12 Evaluation of Filter Theory

9.1.13 End of Topic Test - Sexual Relationships

9.2 Relationships: Romantic Relationships (A2 only)

9.2.1 Social Exchange Theory

9.2.2 Evaluation of Social Exchange Theory

9.2.3 Equity Theory

9.2.4 Evaluation of Equity Theory

9.2.5 Rusbult’s Investment Model

9.2.6 Evaluation of Rusbult's Investment Model

9.2.7 Relationship Breakdown

9.2.8 Studies on Relationship Breakdown

9.2.9 Evaluation of Relationship Breakdown

9.2.10 End of Topic Test - Romantic relationships

9.3 Relationships: Virtual & Parasocial (A2 only)

9.3.1 Virtual Relationships in Social Media

9.3.2 Evaluation of Reduced Cues & Hyperpersonal

9.3.3 Parasocial Relationships

9.3.4 Attachment Theory & Parasocial Relationships

9.3.5 Evaluation of Parasocial Relationship Theories

9.3.6 End of Topic Test - Virtual & Parasocial Realtions

10 Option 1: Gender (A2 only)

10.1 Gender (A2 only)

10.1.1 Sex, Gender & Androgyny

10.1.2 Gender Identity Disorder

10.1.3 Biological & Social Explanations of GID

10.1.4 Biological Influences on Gender

10.1.5 Effects of Hormones on Gender

10.1.6 End of Topic Test - Gender 1

10.1.7 Kohlberg’s Theory of Gender Constancy

10.1.8 Evaluation of Kohlberg's Theory

10.1.9 Gender Schema Theory

10.1.10 Psychodynamic Approach to Gender Development 1

10.1.11 Psychodynamic Approach to Gender Development 2

10.1.12 Social Approach to Gender Development

10.1.13 Criticisms of Social Theory

10.1.14 End of Topic Test - Gender 2

10.1.15 Media Influence on Gender Development

10.1.16 Cross Cultural Research

10.1.17 Childcare & Gender Roles

10.1.18 End of Topic Test - Gender 3

11 Option 1: Cognition & Development (A2 only)

11.1 Cognition & Development (A2 only)

11.1.1 Piaget’s Theory of Cognitive Development 1

11.1.2 Piaget's Theory of Cognitive Development 2

11.1.3 Schema Accommodation Assimilation & Equilibration

11.1.4 Piaget & Inhelder’s Three Mountains Task (1956)

11.1.5 Conservation & Class Inclusion

11.1.6 Evaluation of Piaget

11.1.7 End of Topic Test - Cognition & Development 1

11.1.8 Vygotsky

11.1.9 Evaluation of Vygotsky

11.1.10 Baillargeon

11.1.11 Baillargeon's studies

11.1.12 Evaluation of Baillargeon

11.1.13 End of Topic Test - Cognition & Development 2

11.1.14 Sense of Self & Theory of Mind

11.1.15 Baron-Cohen Studies

11.1.16 Selman’s Five Levels of Perspective Taking

11.1.17 Biological Basis of Social Cognition

11.1.18 Evaluation of Biological Basis of Social Cognition

11.1.19 Important Issues in Social Neuroscience

11.1.20 End of Topic Test - Cognition & Development 3

11.1.21 Top Grade AO2/AO3 - Cognition & Development

12 Option 2: Schizophrenia (A2 only)

12.1 Schizophrenia: Diagnosis (A2 only)

12.1.1 Classification & Diagnosis

12.1.2 Reliability & Validity of Diagnosis

12.1.3 Gender & Cultural Bias

12.1.4 Pinto (2017) & Copeland (1971)

12.1.5 End of Topic Test - Scizophrenia Diagnosis

12.2 Schizophrenia: Treatment (A2 only)

12.2.1 Family-Based Psychological Explanations

12.2.2 Evaluation of Family-Based Explanations

12.2.3 Cognitive Explanations

12.2.4 Drug Therapies

12.2.5 Evaluation of Drug Therapies

12.2.6 Biological Explanations for Schizophrenia

12.2.7 Dopamine Hypothesis

12.2.8 End of Topic Test - Schizoprenia Treatment 1

12.2.9 Psychological Therapies 1

12.2.10 Psychological Therapies 2

12.2.11 Evaluation of Psychological Therapies

12.2.12 Interactionist Approach - Diathesis-Stress Model

12.2.13 Interactionist Approach - Triggers & Treatment

12.2.14 Evaluation of the Interactionist Approach

12.2.15 End of Topic Test - Scizophrenia Treatments 2

13 Option 2: Eating Behaviour (A2 only)

13.1 Eating Behaviour (A2 only)

13.1.1 Explanations for Food Preferences

13.1.2 Birch et al (1987) & Lowe et al (2004)

13.1.3 Control of Eating Behaviours

13.1.4 Control of Eating Behaviour: Leptin

13.1.5 Biological Explanations for Anorexia Nervosa

13.1.6 Psychological Explanations: Family Systems Theory

13.1.7 Psychological Explanations: Social Learning Theory

13.1.8 Psychological Explanations: Cognitive Theory

13.1.9 Biological Explanations for Obesity

13.1.10 Biological Explanations: Studies

13.1.11 Psychological Explanations for Obesity

13.1.12 Psychological Explanations: Studies

13.1.13 End of Topic Test - Eating Behaviour

14 Option 2: Stress (A2 only)

14.1 Stress (A2 only)

14.1.1 Physiology of Stress

14.1.2 Role of Stress in Illness

14.1.3 Role of Stress in Illness: Studies

14.1.4 Social Readjustment Rating Scales

14.1.5 Hassles & Uplifts Scales

14.1.6 Stress, Workload & Control

14.1.7 Stress Level Studies

14.1.8 End of Topic Test - Stress 1

14.1.9 Physiological Measures of Stress

14.1.10 Individual Differences

14.1.11 Stress & Gender

14.1.12 Drug Therapy & Biofeedback for Stress

14.1.13 Stress Inoculation Therapy

14.1.14 Social Support & Stress

14.1.15 End of Topic Test - Stress 2

15 Option 3: Aggression (A2 only)

15.1 Aggression: Physiological (A2 only)

15.1.1 Neural Mechanisms

15.1.2 Serotonin

15.1.3 Hormonal Mechanisms

15.1.4 Genetic Factors

15.1.5 Genetic Factors 2

15.1.6 End of Topic Test - Aggression: Physiological 1

15.1.7 Ethological Explanation

15.1.8 Innate Releasing Mechanisms & Fixed Action Pattern

15.1.9 Evolutionary Explanations

15.1.10 Buss et al (1992) - Sex Differences in Jealousy

15.1.11 Evaluation of Evolutionary Explanations

15.1.12 End of Topic Test - Aggression: Physiological 2

15.2 Aggression: Social Psychological (A2 only)

15.2.1 Social Psychological Explanation

15.2.2 Buss (1963) - Frustration/Aggression

15.2.3 Social Psychological Explanation 2

15.2.4 Social Learning Theory (SLT) 1

15.2.5 Social Learning Theory (SLT) 2

15.2.6 Limitations of Social Learning Theory (SLT)

15.2.7 Deindividuation

15.2.8 Deindividuation 2

15.2.9 Deindividuation - Diener et al (1976)

15.2.10 End of Topic Test - Aggression: Social Psychology

15.2.11 Institutional Aggression: Prisons

15.2.12 Evaluation of Dispositional & Situational

15.2.13 Influence of Computer Games

15.2.14 Influence of Television

15.2.15 Evaluation of Studies on Media

15.2.16 Desensitisation & Disinhibition

15.2.17 Cognitive Priming

15.2.18 End of Topic Test - Aggression: Social Psychology

16 Option 3: Forensic Psychology (A2 only)

16.1 Forensic Psychology (A2 only)

16.1.1 Defining Crime

16.1.2 Measuring Crime

16.1.3 Offender Profiling

16.1.4 Evaluation of Offender Profiling

16.1.5 John Duffy Case Study

16.1.6 Biological Explanations 1

16.1.7 Biological Explanations 2

16.1.8 Evaluation of the Biological Explanation

16.1.9 Cognitive Explanations

16.1.10 Moral Reasoning

16.1.11 Psychodynamic Explanation 1

16.1.12 Psychodynamic Explanation 2

16.1.13 End of Topic Test - Forensic Psychology 1

16.1.14 Differential Association Theory

16.1.15 Custodial Sentencing

16.1.16 Effects of Prison

16.1.17 Evaluation of the Effects of Prison

16.1.18 Recidivism

16.1.19 Behavioural Treatments & Therapies

16.1.20 Effectiveness of Behavioural Treatments

16.1.21 Restorative Justice

16.1.22 End of Topic Test - Forensic Psychology 2

17 Option 3: Addiction (A2 only)

17.1 Addiction (A2 only)

17.1.1 Definition

17.1.2 Brain Neurochemistry Explanation

17.1.3 Learning Theory Explanation

17.1.4 Evaluation of a Learning Theory Explanation

17.1.5 Cognitive Bias

17.1.6 Griffiths on Cognitive Bias

17.1.7 Evaluation of Cognitive Theory (A2 only)

17.1.8 End of Topic Test - Addiction 1

17.1.9 Gambling Addiction & Learning Theory

17.1.10 Social Influences on Addiction 1

17.1.11 Social Influences on Addiction 2

17.1.12 Personal Influences on Addiction

17.1.13 Genetic Explanations of Addiction

17.1.14 End of Topic Test - Addiction 2

17.2 Treating Addiction (A2 only)

17.2.1 Drug Therapy

17.2.2 Behavioural Interventions

17.2.3 Cognitive Behavioural Therapy

17.2.4 Theory of Reasoned Action

17.2.5 Theory of Planned Behaviour

17.2.6 Six Stage Model of Behaviour Change

17.2.7 End of Topic Test - Treating Addiction

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Study Notes

Working Memory Model

Last updated 7 Nov 2023

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Baddeley and Hitch (1974) developed the Working Memory Model (WMM), which focuses specifically on the workings of short-term memory (STM).

Atkinson and Shiffrin’s Multi-Store Model of memory (MSM) was criticised for over-simplifying STM (as well as LTM) as a single storage system, so the WMM alternative proposed that STM is composed of three, limited capacity stores:

• Central Executive – this manages attention, and controls information from the two ‘slave stores’ [below]
• An articulatory rehearsal process (‘inner voice’) of language, including any language presented visually to convert to a phonological state, for storage in the:
• Phonological store (‘inner ear’), which holds auditory speech information and the order in which it was heard (or any visually-presented language converted by the articulatory process)
• Visuo-Spatial Sketchpad – this temporarily retains visual and spatial information

A later addition was the episodic buffer which facilitates communication between the central executive and long term memory.

The three-store STM stemmed from research using a ‘dual-task technique’ (or ‘interference tasks’), whereby performance is measured as participants perform two tasks simultaneously. The following observations provided evidence to suggest different, limited-capacity STM stores process different types of memory:

• If one store is utilised for both tasks, then task performance is poorer than when they are completed separately, due to the store’s limited capacity e.g. repeating “the the the” aloud and reading some text silently would use the articulatory-phonological loop for both tasks, slowing performance.
• If the tasks require different stores, performance would be unaffected when performing them simultaneously e.g. repeating “the the the” aloud whilst performing a reasoning task (requiring attention, i.e. the central executive), or whilst following a mobile stimulus with your eyes (using the visuo-spatial sketchpad).

Evaluation of the Working Memory Model

• The WMM provides an explanation for parallel processing (i.e. where processes involved in a cognitive task occur at once), unlike Atkinson and Shiffrin’s MSM.
• A Shallice and Warrington (1974) case study reported that brain-damaged patient KF could recall verbal but not visual information immediately after its presentation, which supports the WMM’s claim that separate short-term stores manage short-term phonological and visual memories.
• The model was developed based on evidence from laboratory experiments, so confounding variables could be carefully controlled to produce reliable results (that can be replicated).
• Despite providing more detail of STM than the multi-store model, the WMM has been criticized for being too simplistic and vague, e.g. it is unclear what the central executive is, or its exact role in attention.
• Results from laboratory experiments researching the WMM will often have low ecological validity (i.e. may not relate to real life), as tasks such as repeating ‘the the the’ are arguably not representative of our everyday activities.
• Working memory model
• Baddeley and Hitch (1974)

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Up Learn – A Level Psychology (AQA) – Memory

Evaluating the multi-store model of memory: patient kf case study.

The multi-store model predicts that if people have damage to their short-term memory, then they will also have damage to their long-term memory. But patients like patient KF have damage to their short-term memory without damage to their long-term memory. So, the first limitation of the multi-store model is that it isn’t supported by findings from case studies.

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More videos on The Working Memory Model

Introduction (free trial)

Limitations of the Multi-store Model: Patient KF Case Study

Limitations of the Multi-store Model: Short-term Memory Stores (free trial)

Limitations of the Multi-store Model: the Role of Rehearsal (free trial)

Progress Quiz: Limitations of the Multi-store Model (free trial)

The Working Memory Model

Phonological Loop (free trial)

Sub-components of the Phonological Loop (free trial)

Rehearsal and the Word-length Effect (free trial)

Visuo-spatial Sketchpad (free trial)

Sub-components of the Visuo-spatial Sketchpad (free trial)

Episodic Buffer (free trial)

What is Memory?

Types of memory, types of long-term memory, memory accuracy: how good is our memory, exam questions: memory.

Last time we saw the story of patient KF, a man with impaired short-term memory but no damage to his long-term memory, whose condition challenged the multi-store model of memory.

Now, we already saw earlier, that the multi-store model says…

The multi-store model says that we have three memory stores: the sensory register, the short-term memory store, and the long-term memory store.

Information flows unidirectionally through the stores. To pass into short-term memory, information in the sensory register has to be paid attention to.

And to pass into the long-term memory store, information in short-term memory has to be rehearsed.

So, according to the multi-store model, to be stored in long-term memory, information has to first go through short-term memory. If it’s rehearsed, the information is then transferred to long-term memory.

But if it isn’t rehearsed in short-term memory, the information simply fades away.

So, the multi-store model predicts that…

The multi-store model predicts that a person can have damage to their long-term memory without their short-term memory being affected…

…But if a person’s short-term memory is damaged, they lose both their short and their long-term memories… because without being able to rehearse information in short-term memory, it can’t be transferred to long-term memory!

But, we also saw last time that patient KF’s motorbike accident impaired his short-term memory, but left his long-term memory intact.

And later case studies of other patients like KF also revealed similar findings: patients can damage their short-term memory without damaging their long-term memory!

So, a first limitation of the multi-store model is that it isn’t supported by findings from case studies of patients like KF.

Now, we’ll look at two more limitations of the multi-store model next.

But first, to sum up…

To sum it up, the multi-store model predicts that if people have damage to their short-term memory, then they will also have damage to their long-term memory.

But patients like patient KF have damage to their short-term memory without damage to their long-term memory.

So, the first limitation of the multi-store model is that it isn’t supported by findings from case studies.